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1 In contrast, in cancer, where malignant cells exploit heightened proteostasis resilience to tolerate genomic instability and therapeutic stress, strategically destabilizing resilience may enhance therapeutic vulnerability

Considerable reduction in photo system I reaction center subunit IV (Spot n o 17) was observed under HS + AsIII condition, which is in agreement with earlier study showing similar trend 45 irrespective of As stress

While these therapies are still under investigation, early studies in animal models have shown the potential to slow or even reverse some aspects of aging

Results Transcriptional profiling of macrophage sub-populations identifies factors that promote redox diversity and drug tolerance in Mtb We infected primary mouse bone marrow-derived macrophages (BMDMs) with an Mtb strain carrying an established redox biosensor ( Mtb -roGFP2) plasmid that reports E MSH of Mtb 23