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Phospholipase A2 releases AA from cell membranes, which is then metabolized by cytochrome P450 monooxygenases, LOXs, and cyclooxygenases into different products, including hydroperoxides and prostaglandins [38]

Iron deposition not only increases A generation in the brain but also promotes A deposition and oxidative stress (OS)-induced toxicity (Lane et al., 2018

Polysulfide-mediated sulfhydration of sirt1 prevents diabetic nephropathy by suppressing phosphorylation and acetylation of p65 nf-kappab and stat3

This internal defense system is crucial for scavenging free radicals and mitigating oxidative stress throughout the body