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Rapamycin-induced autophagy ameliorates GSH depletion-induced premature cell senescence It was shown that autophagy activation especially in the presence of an increasing autophagic flux inhibits the senescence of RPE cells 35,36,37,38,39

receptor and peroxisome proliferator-activated receptor as integrators of lipid homeostasis and immunity

This influence is primarily mediated by the autonomic nervous system (ANS), which regulates various aspects, including gut permeability, regional gut motility, intestinal transit and secretion

Clinical translation will depend on demonstrating improved diagnostic accuracy, accessibility, and health outcomes across diverse populations