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Funding This work was supported by grants from the National Science and Technology Council of Taiwan (NSTC) 113-2326-B-400-001, 113-2320-B-400-009-MY3 (to J.-J.C.), and 113-2314-B-038-141 (to Y.-T.S.) and the Ministry of Education of Taiwan DP2-TMU-114-C-06 (to Y

Herein, we have demonstrated that CoNPs could induce the ferroptosis-like cell death through the enhancement of intracellular reactive oxygen species (ROS) level, cytoplasmic Fe 2+ level, lipid peroxidation, and consumption of reduced glutathione (GSH) as well as inhibition of glutathione peroxidase 4 (GPX4) activity