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ALC1 Knockdown Enhances Cisplatin Cytotoxicity of Esophageal Squamous Cell Carcinoma Cells by Inhibition of Glycolysis through PI3K/Akt Pathway

A single exposure (A) causes a small decrease (less than 3% of normal), in both blood and cytosolic GSH and Cys concentrations, with levels returning to above 99% of normal for all species within the simulated time-span

Mechanistically distinct from other forms of cell death, cuproptosis is characterized by the direct binding of copper to lipoylated enzymes in the mitochondrial tricarboxylic acid (TCA) cycle, leading to proteotoxic stress and subsequent cell death, independent of classical apoptotic signaling [3]

10.1023/A:1005545012776