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Catecholamines, particularly norepinephrine, are the primary activators of fasting-induced peripheral (adipose) lipolysis, while cortisol, glucagon, growth hormone (GH), and adrenocorticotropic hormone (ACTH) also activate lipolysis [178]

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This analysis encompassed the impact of oxidative stress on VSMCs, ECM, inflammatory cells, and endothelial cells

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