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The as-prepared LGNCs exhibited excellent PL properties, which can be further enhanced by the peptide-induced NC aggregation

N-acetylcysteine can reduce the disulfide bonds in a dose-dependent manner, 41,42 decreasing angiotensin II and increasing angiotensin 17 (a biologically active peptide exerting many opposing actions to angiotensin II), thus protecting against lung inflammation and fibrosis