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Biochemical evaluation of oxidative stress in propylthiouracil treated hyperthyroid patients

The lysates were pre-cleared by incubation with biotin-blocked streptavidin-conjugated magnetic beads

Generation of cellular ROS production, decrease in nitric oxide bioavailability, and activation of enzymes like NADPH oxidase, and many signaling factors induce oxidative stress followed by breakdown of redox balance with consequential development of physiological and metabolic disorders, including endothelial dysfunction, inflammation and increasing concentration of VSMCs

Biochim Biophys Acta 1270, 173178.Google ScholarPubMed in vivo : studies upon the profile of polyunsaturated fatty acids