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Specific and moderate manipulation of activities of LOXs and transcriptional regulation of hypoxia-responsive transcription factors through copper bioavailability modulation or continuous hypoxia-conditioning may become effective interventions for enhanced cartilage regeneration, as well as promising rejuvenation therapeutics, which may exert further therapeutic implications in the upcoming clinical arena

Autophagy and ferroptosis involve complex metabolic interactions: autophagy-dependent degradation of ferritin leads to the accumulation of free iron and promotes lipid peroxidation through the Fenton reaction

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