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The selectivity profile confirmed in multiple rodent and early human studies allows researchers to attribute observed experimental outcomes specifically to GH-axis modulation rather than to concurrent adrenal or lactotroph activation, a significant methodological advantage over the hexapeptide GHRPs

In clinical populations, approaches combining cortical thickness (structural MRI), amyloid-PET, tau-PET, and white matter lesion burden may predict domain-specific cognitive decline with precision [601, 602]